دورية أكاديمية
Enriching Islet Phospholipids With Eicosapentaenoic Acid Reduces Prostaglandin E 2 Signaling and Enhances Diabetic β-Cell Function.
العنوان: | Enriching Islet Phospholipids With Eicosapentaenoic Acid Reduces Prostaglandin E |
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المؤلفون: | Neuman JC; Interdisciplinary Graduate Program in Nutritional Sciences, College of Agriculture and Life Sciences, University of Wisconsin-Madison, Madison, WI.; Research Service, William S. Middleton Memorial Veterans Hospital, Madison, WI., Schaid MD; Interdisciplinary Graduate Program in Nutritional Sciences, College of Agriculture and Life Sciences, University of Wisconsin-Madison, Madison, WI.; Research Service, William S. Middleton Memorial Veterans Hospital, Madison, WI., Brill AL; Research Service, William S. Middleton Memorial Veterans Hospital, Madison, WI.; Department of Medicine, Division of Endocrinology, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI., Fenske RJ; Interdisciplinary Graduate Program in Nutritional Sciences, College of Agriculture and Life Sciences, University of Wisconsin-Madison, Madison, WI.; Research Service, William S. Middleton Memorial Veterans Hospital, Madison, WI., Kibbe CR; Research Service, William S. Middleton Memorial Veterans Hospital, Madison, WI.; Department of Medicine, Division of Endocrinology, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI., Fontaine DA; Research Service, William S. Middleton Memorial Veterans Hospital, Madison, WI.; Department of Medicine, Division of Endocrinology, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI., Sdao SM; Research Service, William S. Middleton Memorial Veterans Hospital, Madison, WI.; Integrated Program in Biochemistry, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI., Brar HK; Research Service, William S. Middleton Memorial Veterans Hospital, Madison, WI.; Department of Medicine, Division of Endocrinology, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI., Connors KM; Research Service, William S. Middleton Memorial Veterans Hospital, Madison, WI.; Department of Medicine, Division of Endocrinology, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI., Wienkes HN; Research Service, William S. Middleton Memorial Veterans Hospital, Madison, WI.; Department of Medicine, Division of Endocrinology, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI., Eliceiri KW; Department of Biomedical Engineering, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI.; Laboratory for Optical and Computational Instrumentation, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI., Merrins MJ; Research Service, William S. Middleton Memorial Veterans Hospital, Madison, WI.; Department of Medicine, Division of Endocrinology, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI.; Integrated Program in Biochemistry, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI.; Laboratory for Optical and Computational Instrumentation, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI.; Department of Biomolecular Chemistry, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI., Davis DB; Interdisciplinary Graduate Program in Nutritional Sciences, College of Agriculture and Life Sciences, University of Wisconsin-Madison, Madison, WI.; Department of Medicine, Division of Endocrinology, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI.; Medical Service, William S. Middleton Memorial Veterans Hospital, Madison, WI., Kimple ME; Interdisciplinary Graduate Program in Nutritional Sciences, College of Agriculture and Life Sciences, University of Wisconsin-Madison, Madison, WI mkimple@medicine.wisc.edu.; Research Service, William S. Middleton Memorial Veterans Hospital, Madison, WI.; Department of Medicine, Division of Endocrinology, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI.; Department of Cell and Regenerative Biology, University of Wisconsin-Madison School of Medicine and Public Health, Madison, WI. |
المصدر: | Diabetes [Diabetes] 2017 Jun; Vol. 66 (6), pp. 1572-1585. Date of Electronic Publication: 2017 Feb 13. |
نوع المنشور: | Journal Article |
اللغة: | English |
بيانات الدورية: | Publisher: American Diabetes Association Country of Publication: United States NLM ID: 0372763 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1939-327X (Electronic) Linking ISSN: 00121797 NLM ISO Abbreviation: Diabetes Subsets: MEDLINE |
أسماء مطبوعة: | Publication: Alexandria, VA : American Diabetes Association Original Publication: [New York, American Diabetes Association] |
مواضيع طبية MeSH: | Alprostadil/*analogs & derivatives , Diabetes Mellitus/*metabolism , Dinoprostone/*metabolism , Eicosapentaenoic Acid/*pharmacology , Glucose/*metabolism , Insulin/*metabolism , Insulin-Secreting Cells/*drug effects , Receptors, Prostaglandin E, EP3 Subtype/*drug effects, Alprostadil/metabolism ; Animals ; Arachidonic Acid/metabolism ; Chromatography, Gas ; Gene Expression Profiling ; Insulin Secretion ; Insulin-Secreting Cells/metabolism ; Interleukin-1beta/pharmacology ; Islets of Langerhans/drug effects ; Islets of Langerhans/metabolism ; Mass Spectrometry ; Mice ; Mice, Inbred NOD ; Mice, Obese ; Optical Imaging ; Phospholipids ; Receptors, Prostaglandin E, EP3 Subtype/metabolism ; Signal Transduction |
مستخلص: | Prostaglandin E (© 2017 by the American Diabetes Association.) |
التعليقات: | Comment in: Diabetes. 2017 Jun;66(6):1464-1466. (PMID: 28533298) |
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معلومات مُعتمدة: | R01 DK110324 United States DK NIDDK NIH HHS; R01 DK113103 United States DK NIDDK NIH HHS; R21 AG050135 United States AG NIA NIH HHS; K01 DK101683 United States DK NIDDK NIH HHS; T32 AG000213 United States AG NIA NIH HHS; R01 DK102598 United States DK NIDDK NIH HHS; I01 BX001880 United States BX BLRD VA; P50 AG033514 United States AG NIA NIH HHS |
المشرفين على المادة: | 0 (Insulin) 0 (Interleukin-1beta) 0 (Phospholipids) 0 (Ptger3 protein, mouse) 0 (Receptors, Prostaglandin E, EP3 Subtype) 27YG812J1I (Arachidonic Acid) 802-31-3 (prostaglandin E3) AAN7QOV9EA (Eicosapentaenoic Acid) F5TD010360 (Alprostadil) IY9XDZ35W2 (Glucose) K7Q1JQR04M (Dinoprostone) |
تواريخ الأحداث: | Date Created: 20170215 Date Completed: 20170822 Latest Revision: 20210305 |
رمز التحديث: | 20221213 |
مُعرف محوري في PubMed: | PMC5440023 |
DOI: | 10.2337/db16-1362 |
PMID: | 28193789 |
قاعدة البيانات: | MEDLINE |
تدمد: | 1939-327X |
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DOI: | 10.2337/db16-1362 |