دورية أكاديمية
Molecular mechanisms of kidney crosstalk with distant organs.
العنوان: | Molecular mechanisms of kidney crosstalk with distant organs. |
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المؤلفون: | Hasegawa S; Division of CKD Pathophysiology, The University of Tokyo Graduate School of Medicine, Tokyo, Japan.; Division of Nephrology and Endocrinology, The University of Tokyo Graduate School of Medicine, Tokyo, Japan., Inagi R; Division of CKD Pathophysiology, The University of Tokyo Graduate School of Medicine, Tokyo, Japan. inagi-r@m.u-tokyo.ac.jp. |
المصدر: | Nature reviews. Nephrology [Nat Rev Nephrol] 2023 Feb; Vol. 19 (2), pp. 75-76. |
نوع المنشور: | Journal Article |
اللغة: | English |
بيانات الدورية: | Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101500081 Publication Model: Print Cited Medium: Internet ISSN: 1759-507X (Electronic) Linking ISSN: 17595061 NLM ISO Abbreviation: Nat Rev Nephrol Subsets: MEDLINE |
أسماء مطبوعة: | Original Publication: London Nature Pub. Group |
مواضيع طبية MeSH: | Kidney* , Acute Kidney Injury*, Humans |
References: | Khamissi, F. Z. et al. Identification of kidney injury released circulating osteopontin as causal agent of respiratory failure. Sci. Adv. 8, eabm5900 (2022). (PMID: 10.1126/sciadv.abm5900) Hüsing, A. M. et al. Myeloid CCR2 promotes atherosclerosis after AKI. J. Am. Soc. Nephrol. 33, 1487–1500 (2022). (PMID: 10.1681/ASN.2022010048) Arinze, N. V. et al. Tryptophan metabolites suppress the Wnt pathway and promote adverse limb events in chronic kidney disease. J. Clin. Invest. 132, ••• (2022). Linh, H. T. et al. Intestinal bacterial translocation contributes to diabetic kidney disease. J. Am. Soc. Nephrol. 33, 1105–1119 (2022). (PMID: 10.1681/ASN.2021060843) Egstrand, S. et al. Hypomorphic expression of parathyroid Bmal1 disrupts the internal parathyroid circadian clock and increases parathyroid cell proliferation in response to uremia. Kidney Int. 101, 1232–1250 (2022). (PMID: 10.1016/j.kint.2022.02.018) Hasegawa, S. et al. Activation of sympathetic signaling in macrophages blocks systemic inflammation and protects against renal ischemia-reperfusion injury. J. Am. Soc. Nephrol. 32, 1599–1615 (2021). (PMID: 10.1681/ASN.2020121723) Salyers, Z. R., Coleman, M., Balestrieri, N. P. & Ryan, T. E. Indoxyl sulfate impairs angiogenesis via chronic aryl hydrocarbon receptor activation. Am. J. Physiol. Cell Physiol. 320, C240–C249 (2021). (PMID: 10.1152/ajpcell.00262.2020) Yang, J. et al. Intestinal microbiota control acute kidney injury severity by immune modulation. Kidney Int. 98, 932–946 (2020). (PMID: 10.1016/j.kint.2020.04.048) Motohashi, H. et al. The circadian clock is disrupted in mice with adenine-induced tubulointerstitial nephropathy. Kidney Int. 97, 728–740 (2020). (PMID: 10.1016/j.kint.2019.09.032) Yoshida, Y. et al. Alteration of circadian machinery in monocytes underlies chronic kidney disease-associated cardiac inflammation and fibrosis. Nat. Commun. 12, 2783 (2021). (PMID: 10.1038/s41467-021-23050-x) |
تواريخ الأحداث: | Date Created: 20221126 Date Completed: 20230129 Latest Revision: 20230201 |
رمز التحديث: | 20240628 |
DOI: | 10.1038/s41581-022-00655-z |
PMID: | 36434161 |
قاعدة البيانات: | MEDLINE |
تدمد: | 1759-507X |
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DOI: | 10.1038/s41581-022-00655-z |