دورية أكاديمية
COVID-19-induced cardiovascular damage differs from other prevalent viruses
العنوان: | COVID-19-induced cardiovascular damage differs from other prevalent viruses |
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المؤلفون: | Rachel S Parise, Sindhu Ramesh, Manoj Govindarajulu, Amir Ajoolabady, Timothy Moore, Muralikrishnan Dhanasekaran |
المصدر: | Cardiology Plus, Vol 6, Iss 4, Pp 231-245 (2021) |
بيانات النشر: | Wolters Kluwer Health/LWW, 2021. |
سنة النشر: | 2021 |
المجموعة: | LCC:Diseases of the circulatory (Cardiovascular) system |
مصطلحات موضوعية: | cardiovascular disease, cardiovascular pathology, covid-19, heart damage, severe acute respiratory syndrome-coronavirus-1, Diseases of the circulatory (Cardiovascular) system, RC666-701 |
الوصف: | Viral infections persist globally, among all ages, gender, and ethnicity. Of particular importance is COVID-19, associated with asymptomatic to severe symptoms, including complications/mortality. Cardiovascular disease (CVD) involves heart and blood vessel disorders including coronary heart disease, cerebrovascular disease, peripheral artery disease, thrombosis, and more. CVD associated with severe COVID-19 includes heart failure, coronary artery disease, cardiomyopathy, hypertension, and cerebrovascular disease/stroke. Data were acquired from PubMed, Google Scholar, Centers for Disease Prevention and Control, and Lexi-Comp using the search terms “COVID-19 and cardiovascular pathology;” “COVID-19 induced CVD;” “Viral infection induced CVD;” and “Viral infection induced heart damage.” COVID-19-induced CVD mechanisms include direct viral entry, inflammation, cytokine storm, hypoxia, interferon-mediated immune response, plaque destabilization, stress, and drug-induced causes. Other viral pathologies causing CVD include atherosclerosis, inflammation, cytokine storm, and plaque destabilization. Individual parameters, such as old age, males, and higher body mass index (BMI), are more likely to experience viral-associated complications, possibly explained by patient risk factors or comorbidities. Populations at higher risk include older males with an elevated BMI. Viral mechanisms associated with CVD are similar but differ in disease severity, potentially explained by diverse cytokine profiles where COVID-19 activates different types at higher quantities. |
نوع الوثيقة: | article |
وصف الملف: | electronic resource |
اللغة: | English |
تدمد: | 2470-7511 2470-752X |
Relation: | http://www.cardiologyplus.org/article.asp?issn=2470-7511;year=2021;volume=6;issue=4;spage=231;epage=245;aulast=Parise; https://doaj.org/toc/2470-7511; https://doaj.org/toc/2470-752X |
DOI: | 10.4103/2470-7511.334401 |
URL الوصول: | https://doaj.org/article/089c5b0abad44ae18280e91992dc68f8 |
رقم الأكسشن: | edsdoj.089c5b0abad44ae18280e91992dc68f8 |
قاعدة البيانات: | Directory of Open Access Journals |
تدمد: | 24707511 2470752X |
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DOI: | 10.4103/2470-7511.334401 |