التفاصيل البيبلوغرافية
العنوان:
What drives progressive motor deficits in patients with acute pontine infarction?
المؤلفون:
Jue-bao Li , Rui-dong Cheng , Liang Zhou , Wan-shun Wen , Gen-ying Zhu , Liang Tian , Xiang-ming Ye
المصدر:
Neural Regeneration Research, Vol 10, Iss 3, Pp 501-504 (2015)
بيانات النشر:
Wolters Kluwer Medknow Publications, 2015.
سنة النشر:
2015
المجموعة:
LCC:Neurology. Diseases of the nervous system
مصطلحات موضوعية:
tamoxifen , Src kinase , PP2 , trauma , regeneration , neuroprotection , auranofin , dextromethorphan , rosiglitazone , Alzheimer′s disease , neuroinflammation , neurodegeneration , microglia , astrocytes , nerve regeneration , spinal cord , electroacupuncture therapy , neural stem cells , notch signaling pathway , inflammation , survival curve , proliferation , differentiation , real-time quantitative PCR , western blot assay , neural regeneration , superparamagnetic iron oxide , magnetic guidance , bone marrow mesenchymal stem cells , spinal cord injury , cell transplantation , magnetic resonance image , lumbar puncture , spinal cord transection , average combined score , magnetic resonance imaging , diffusion tensor imaging , fractional anisotropy , apparent diffusion coefficient , fiber tractography , peripheral nerve injury , sciatic nerve , hypothermia , blood-nerve barrier , Evans blue tracer , neural degeneration , polyethyleneimine-polyethylene glycol , spiral ganglion cells , X-linked inhibitor of apoptosis protein , gene therapy , nanocarrier , cisplatin , ototoxicity , cochlea , ocular hypertension , JNK3 , retinal ganglion cell , glaucoma , laser photocoagulation , intraocular pressure , brain injury , apoptosis , cerebral ischemia , SMAD3 , transforming growth factor β1 , NSFC grant , Chinese herbal formula , Tneurotrophic factor , ongluo Jiunao injection , nerve growth factor receptor , Xuesai Tong , hippocampus , dentate gyrus , lipid peroxidation , type 1 diabetes , malondialdehyde , neurons , acupuncture , cerebral hemorrhage , immunohistochemistry , Notch1 , Hes1 , rats , DAPT , transcranial direct current stimulation , visuomotor coordination , task difficulty , primary motor area , motor learning , paraquat , poisoning , endoplasmic reticulum stress , ulinastatin , CHOP , GRP78 , caspase-3 , reactive oxygen species , cognitive neuroscience , event-related potential , P300 , electrophysiology , nutritional state , minimally conscious state , consciousness , disorders of consciousness , unconsciousness , evaluation , prognosis , NSFC grants , curcumin , ischemia/reperfusion injury , oxidative stress , primary cell culture , cortical neurons , oxygen-glucose deprivation , pretreatment , post-treatment , blood oxygen level-dependent functional magnetic resonance imaging , dysphagia , function restructuring , cerebral cortex , pontine infarction , progressive motor deficits , basilar artery , penetrating artery , corticospinal tract , Wallerian degeneration , review , Neurology. Diseases of the nervous system , RC346-429
الوصف:
Progressive motor deficits are relatively common in acute pontine infarction and frequently associated with increased functional disability. However, the factors that affect the progression of clinical motor weakness are largely unknown. Previous studies have suggested that pontine infarctions are caused mainly by basilar artery stenosis and penetrating artery disease. Recently, lower pons lesions in patients with acute pontine infarctions have been reported to be related to progressive motor deficits, and ensuing that damage to the corticospinal tracts may be responsible for the worsening of neurological symptoms. Here, we review studies on motor weakness progression in pontine infarction and discuss the mechanisms that may underlie the neurologic worsening.
نوع الوثيقة:
article
وصف الملف:
electronic resource
اللغة:
English
تدمد:
1673-5374
Relation:
http://www.nrronline.org/article.asp?issn=1673-5374;year=2015;volume=10;issue=3;spage=501;epage=504;aulast=Li ; https://doaj.org/toc/1673-5374
DOI:
10.4103/1673-5374.153703
URL الوصول:
https://doaj.org/article/3df0ef4f34a3458ab7626c378c4913d9
رقم الأكسشن:
edsdoj.3df0ef4f34a3458ab7626c378c4913d9
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